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Submitted: 15 Dec 2023
Accepted: 01 Dec 2024
ePublished: 28 Dec 2024
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J Nephropharmacol. 2025;14(1): e11667.
doi: 10.34172/npj.2025.11667
  Abstract View: 1
  PDF Download: 3

Clinical Trial

Effect of atorvastatin on pro-inflammatory cytokines during cisplatin administration; a double-blind clinical trial study

Seyedeh Azra Shamsdin 1 ORCID logo, Nasrin Namdari 2* ORCID logo, Ali Rajaei 3

1 Gastroenterohepatology Research Centre, Shiraz University of Medical Science, Shiraz, Iran
2 Hematology and Medical Oncology Department, Shiraz University of Medical Science, Shiraz, Iran
3 Internal Medicine Department, Shiraz University of Medical Science, Shiraz, Iran
*Corresponding Author: Nasrin Namdari, Email: Namdari_N@sums.ac.ir, Email: Sonanamdari@yahoo.com

Abstract

Introduction: The primary concern in maintaining treatment with cisplatin pertains to the occurrence of cisplatin-induced nephrotoxicity. After the administration of cisplatin, the prospective evaluation of the effects of Atorvastatin on kidney function and inflammatory mediators was conducted.

Objectives: This double-blind clinical trial study was conducted in order to assess the changes in serum levels of tumor necrosis factor alpha (TNF-α)and interleukin-17A (IL-17A) after cisplatin administration and to ascertain the protective effects of atorvastatin against cisplatin nephrotoxicity due to the effect on the levels of cytokines.

Patients and Methods: In this double-blind clinical trial study, 30 potential candidates for the administration of cisplatin were enrolled. Patients were subsequently categorized into two distinct groups. Group A received 40 mg/d of atorvastatin on the first day of cisplatin and continued for seven days. Group B was provided with a placebo. Following the administration of cisplatin, blood samples were collected for BUN, serum creatinine, magnesium, potassium, TNF-α, and IL-17A on days 0, 8 and 21.

Results: Although there was no statistically significant difference in terms of serum urea, creatinine and glomerular filtration rate (GFR) between the two groups; however, within intra-group analysis, atorvastatin caused slower reduction of GFR compared to baseline. Though, no correlation was observed between the level of inflammatory cytokines including TNF-α and IL-17A and atorvastatin administration.

Conclusion: Atorvastatin may have the ability to prevent cisplatin nephrotoxicity; however, it does not have any effect on inflammatory cytokines.

Trial Registration: The trial protocol was approved by the Iranian Registry of Clinical Trial (identifier: IRCT20140605017982N2; https://en.irct.ir/trial/47349, ethical code: IR.SUMS.MED. REC.1398.024).



Implication for health policy/practice/research/medical education:

The nephrotoxicity of cisplatin is a significant constraint on the use of drugs. With effective preventive measures, oncologists should be able to use the drug safely. By prescribing atorvastatin before cisplatin, a smaller drop-in glomerular filtration rate occurs, which seems can be used to prevent kidney injury by cisplatin, although no significant relationship was found with the level of inflammatory cytokines, which may be due to a different mechanism of atorvastatin.

Please cite this paper as: Shamsdin SA, Namdari N, Rajae A. Effect of atorvastatin on pro-inflammatory cytokines during cisplatin administration; a double-blind clinical trial study. J Nephropharmacol. 2025;14(1):e11667. DOI: 10.34172/npj.2025.11667.

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